Digestive Health Treatment
Find answers and solutions to Digestive challenges/ health at the Get Well Center:
If you or a loved one is suffering from a gastrointestinal problem, The Get Well Center will listen and asses you, diagnose the underlying cause of your health concerns, then administer care that's effective & natural to restore your health and vitality.
Digestive (Gastrointestinal) Health Concerns:
This may require specific tests for advanced diagnosis like, extensive digestive studies, appropriate blood studies, X-ray, MRI, CT, Applied Kinesiology, stool, urine, and saliva studies. This will help assure the most effective care to resolve your underlining problem.
The Get Well Center provides a wide range of natural care like: cleanses, Dr. managed diets, individualized nutritional support and repair, cleanses, probiotics, digestive enzymes, herbal and homeopathic remedies, acupuncture, etc.
If your tired of feeling sick and tired, your worth a healthier tomorrow, get started on the path to Getting Well!
Answers and alternative care for digestive difficulties such as:
• Undigested food
• Diarrhea (loose stool)
• Explosive stool
• Indigestion (Dyspepsia)
• Excessive gas, Foul Gas
• Stomach or Abdominal pain, cramping, ache, or weakness
• Other symptoms related to digestive problems
• What is NORMAL bowel movement?
Diagnosed with a gastrointestinal condition and looking for answers?
• Gluten Sensitivity
• Celiac’s (Coeliac) Disease or Sprue
• Acid Reflux Disease (GERD)
• Irritable Bowel Syndrome (IBS)
• Hiatal Hernia
• Inflammatory bowel disease (IBD) • Leaky Gut
• Yeast or Candida Overgrowth
• Ulcerative Colitis
• Intestinal Parasites
• Crohn’s Disease
• Dysmbiosis (Gut bacteria imbalance)
• Peptic Ulcer
• Food Sensitivity
• Food Allergy
Did you know that the following symptoms may also be related to a gastrointestinal problem?
Headaches, fatigue, poor sleep, bad mood, depression, general muscle and joint aches and stiffness, auto immune disease, attention deficit or poor concentration, or poor memory.
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Normal Bowel Movement:
One should have 1 to 2 bowel movements per day and the consistency of the stool should be like peanut butter with-out splattering around the bowl.
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Diarrhea (loose stools):
Diarrhea is when the bowel movement is runny or watery. When this occurs one or more times a week there is a problem with your digestion.
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Constipation is when the stools are hard, dry, black like tare, or there is difficulty in emptying the stool. Also, you are constipated when you have a bowel movement less than once a day or the bowel movement is incomplete.
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Indigestion or Dyspepsia
Indigestion refers to a feeling of fullness or discomfort during or after a meal. Other symptoms often experienced with indigestion are; burning or pain in the upper stomach, belching, nausea, vomiting, acid taste, and growling stomach. These symptoms may increase in times of stress.
Indigestion is usually not a condition on its own, indigestion’s likely a sign of an underlying problem, such as gastroesophageal reflux disease (GERD), ulcers, or gallbladder disease.
People often have heartburn (a burning sensation deep in the chest) along with indigestion. But heartburn is caused by stomach acids rising into the esophagus.
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Heartburn or pyrosis is a painful and burning sensation in the esophagus, just below the breastbone typically associated with regurgitation of stomach acid. The burning pain often ascends into the chest and may climb up to the neck or throat. Despite its name, heartburn has nothing to do with the heart, although some heart problems do have a similar sensation to heartburn.
Note, that chronic reoccurring heartburn results in acid reflux disease or GERD
A restricting diet is very important, since approximately 90-95% of sufferers of heartburn or esophageal disorder can link their symptoms to specific foods. Therefore, it’s important that sufferers of heartburn manage their diets as a way to cope with their heartburn. Specifically, it has been shown that fatty foods and caffeinated beverages can cause the symptoms of heartburn.
Other self-management includes raising one's sleeping bed at a slight angle so that the head is raised slightly higher than the feet. This small angle is intended to prevent stomach acid from rising in the esophagus and causing pain.
Be aware that these are approaches are what one can do at home to help alleviate the condition, not correct the cause. Getting proper diagnosis and natural care to remedy or help correct the underlying condition is what The Get Well Center is about.
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When you see particles of undigested food in your stool this indicates the inability to digest your food completely, a sign of food sensitivity, allergy or malabsorption thus malnourishment may be present.
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Malabsorption occurs whit the digestive track’s inability to break down the foods (proteins, fats, carbs) or absorb the nutrients / building blocks necessary for proper health. Without the proper nutrients the body can not properly grow, develop and repair itself thus, poor health develops which opens the door to disease.
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Gluten is a composite of the protein’s gliadin and glutenin. Gluten Sensitive is when one has adverse reaction like bloating, gas, or fatigue, when eating gluten. To avoid one’s reaction to gluten one must eat a diet completely free of ingredients that contain gluten. Gluten is found in wheat (including spelt and kamut), barley, triticale, rye, and oats. Gluten is notoriously found in products like flour, pasta, bakery, pastries, bread, cereals, food additives, stabilizing agent or thickener in products like ice-cream and ketchup and mash in beer. Read labels if on gluten free diet.
Gluten free can be found in several grains and starch sources and are considered acceptable for a gluten-sensitive diet. The most frequently used are maize, potatoes, rice, and tapioca (derived from cassava). Other grains and starch sources generally considered suitable for gluten-free diets include amaranth, arrowroot, millet, montina, lupine, quinoa, sorghum (jowar), sweet potato, taro, teff, chia seed, and yam. Various types of bean, soybean, and nut flours are sometimes used in gluten-free products to add protein and dietary fiber. In spite of its name, buckwheat is not related to wheat; pure buckwheat is considered acceptable for a gluten-free diet, although many commercial buckwheat products are actually mixtures of wheat and buckwheat flours, and thus not acceptable. Gram flour, derived from chickpeas, is also gluten-free.
The suitability of oats in the gluten free diet is still controversial. Recent research indicated that a protein naturally found in oats (avenin) possessed peptide sequences closely resembling wheat gluten and caused mucosal inflammation in significant numbers (about 10%). In addition, the processing of oats are virtually always contaminated by other grains that contain gluten thus most gluten sufferers are sensitive to oats.
Gluten free can also be found with highly processed wheat in which the gluten proteins gliadin and glutenin are extracted out leaving only the wheat starch (found to contain no detectable gluten is less than 5 parts per million).
The stored proteins of maize and rice are sometimes called glutens, but their proteins differ from wheat gluten by lacking gliadin.
Gluten and Alcoholic beverages
Almost all beers are brewed with malted barley or wheat and will contain gluten. Sorghum and buckwheat-based gluten-free beers are available but remain very much a specialty product.
Although most forms of whiskey are distilled from a mash (includes grains that contain gluten), distillation removes any proteins present in the mash, including gluten. Spirits made without any grain such as brandy, wine, mead, cider, sherry, port, rum, tequila and vermouth do not contain gluten, although some vineyards use a flour paste to caulk the oak barrels in which wine is aged.
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Celiac’s disease or Coeliac disease
Is an autoimmune disorder where gluten causes an abnormal immune reaction that results in the atrophy the villus of the small intestine. In other words, your own body attacks the small intestine causing in destruction of the intestinal lining and resulting in the in-ability to absorb nutrients for nutrition.
By definition, a diagnosis of Celiac Disease requires biopsy evidence of intestinal villous atrophy. Gluten sensitivity refers to those who have an abnormal immune response to dietary gluten, but may not show biopsy evidence of villous atrophy.
This condition has several other names, including: cœliac disease, c(o)eliac sprue, non-tropical sprue, endemic sprue, gluten enteropathy or gluten-sensitive enteropathy, and gluten intolerance.
Celiac’s disease occurs in genetically predispose people of all ages, as many as 1 in 100 Americans has celiac disease and a growing portion of diagnoses are being made in asymptomatic persons as a result of increased screening. The manifestations of celiac disease range from no symptoms to malabsorption of nutrients with involvement of multiple organ systems. The only effective treatment is a lifelong gluten-free diet.
Symptoms of coeliac disease may include abdominal bloating, abdominal pain, mouth ulcers, nausea, vomiting, diarrhea, weight loss (or stunted growth in children), and fatigue.
Celiac’s disease leads to malabsorption as the bowel becomes damaged which may lead to hyperparathyroidism, osteopenia, osteoporosis, muscle loss, psychosocial problems, lactose intolerance, and anemia.
Coeliac disease leads to an increased risk of both adenocarcinoma and lymphoma of the small bowel, which returns to baseline with diet. Longstanding disease may lead to other complications, such as ulcerative jejunitis (ulcer formation of the small bowel) and stricturing (narrowing as a result of scarring).
Other associated diseases to ceoliac’s is the autoimmune disorders: diabetes mellitus type 1, autoimmune thyroiditis, primary biliary cirrhosis, autoimmune hepatitis and microscopic colitis.
Both Celiac Disease and Gluten Sensitivity involve an IgA and/or IgG immune system response to dietary gluten. This differs from classic allergy, which involves an IgE response. In Celiac Disease, autoantibodies (anti-tTG, anti-endomysial) are also involved that attack the intestinal villi.
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Acid Reflux Disease or GERD
Gastroesophageal reflux disease (GERD) or Acid reflux disease is when heartburn occurs over long periods of time, resulting in damage to the lining of the esophagus (mucosal).
Acid reflux disease usually occurs with changes at the junction of the esophagus and the stomach. This can be due to a weak lower esophageal sphincter, hiatal hernia, or abnormal stomach acid.
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Irritable Bowel Syndrome or IBS:
IBS also known as Spastic Colon is a general term used to cover a variety functional bowel disorder characterized by chronic abdominal pain, discomfort, bloating, and abnormal bowel habits. In some cases, one finds their symptoms to be relieved by bowel movements. Diarrhea or constipation may predominate, or they may alternate. IBS may begin after an infection or a stressful life event.
It is important to not misdiagnose because there are several other conditions in which symptoms may emulate IBS such as: coeliac disease, mild infections, parasitic infections like giardiasis, helicobacter pylori, lactose intolerance, food allergies, and several inflammatory bowel diseases. In IBS, basic clinical tests yield no abnormalities. The exact cause of IBS is unknown. The most common theory is that IBS is a disorder of the interaction between the brain and the gastrointestinal tract, although there may also be abnormalities in the gut flora or the immune system. In most patients IBS does not lead to more serious conditions. But it is a source of chronic pain, fatigue and other symptoms, and it increases a patient's medical costs, and contributes to work absenteeism thus, can dramatically affect the quality of a sufferer's life.
The Get Well Center recommends that all patients with symptoms of IBS be tested for celiac disease. Chronic use of certain sedative-hypnotic drugs especially the benzodiazepines may cause irritable bowel like symptoms which can lead to a misdiagnosis of irritable bowel syndrome.
A 2008 review has found probiotics to be beneficial in the treatment of IBS. Many different types have been found to be effective including: Lactobacillus plantarum and Bifidobacteria infantis; however, one review found that only Bifidobacteria infantis showed efficacy.
There is a strong brain-gut component to IBS. Relaxation therapy has also been found to helpful.
The multi-herbal extract Iberogast was found to be significantly superior to placebo via both an abdominal pain scale and an IBS symptom score after four weeks of treatment.
Enteric coated peppermint oil capsules has been advocated for IBS symptoms in adults and children; however, results from trials have been inconsistent.
Many sufferers of IBS seek relief using acupuncture.
At The Get Well Center we take into consideration all that is discussed above and investigate the condition of your health to discover the possible underlying causes of your symptoms. Some of the possible underlying gastro-intestinal condition are: yeast overgrowth, food sensitivity, abnormal gut flora, parasitic, neurological hypo/hypertonicity…
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Colitis is a general term describing chronic digestive disease that contains an inflammation and auto-immune component to the tissue lining the gastrointestinal track (large and small intestine). It is classed as an inflammatory bowel disease (IBD), not to be confused with irritable bowel syndrome (IBS).
Types of colitis include ulcerative colitis (UC), Crohn's colitis, diversion colitis, ischemic colitis, infectious colitis, fulminant colitis, collagenous colitis, chemical colitis, microscopic colitis, lymphocytic colitis, and atypical colitis.
Signs and symptoms
General symptoms of colitis include pain, tenderness in the abdomen, depression, rapid weight loss, aches and pains within the joints, fatigue, fever, changes in bowel habits (increased frequency), and diarrhea may present itself, although some forms of colitis are constipation so, the stool and bowel movements can appear normal. Other symptoms may include: gas, bloating, indigestion, heartburn, reflux, Gastro esophageal reflux disease (GERD), cramps, urgency and many other uncomfortable aches in the gastrointestinal system.
Signs include swelling of the colon, erythema (redness) of the surface of the colon, ulcers on the colon (in ulcerative colitis) which can bleed, mucus in the stool, blood in stool and rectal bleeding.
Common tests which reveal these signs include X-rays of the colon, testing the stool for blood and pus, sigmoidoscopy, and colonoscopy. Additional tests include stool cultures and blood tests, including blood chemistry tests. A high erythrocyte sedimentation rate (ESR) is one typical finding in acute exacerbations of colitis.
Many people have found that one or more of the following foods can trigger their symptoms:
• carbonated beverages
• dairy products, if lactose intolerant
• dried beans, peas, and legumes
• dried fruits, berries, fruits with pulp or seeds
• foods containing sulfur or sulfate
• foods high in fiber, including whole-grain products
• hot sauce, pepper
• nuts, crunchy nut butters
• products containing sorbitol (sugar-free gum and candies)
• raw vegetables
• refined sugar
• spicy foods, sauces
They’re many incidents were people with colitis claim they have become completely symptom free by eliminating most foods, especially grains and other cooked foods, and replacing them with a raw food diet based around fruits.
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Ulcerative colitis is a form of inflammatory bowel disease (IBD) and colitis It’s characterized by ulcers, or open sores, in the colon. Symptoms of ulcerative colitis depends on the severity of the disease process. Patients usually present with diarrhea mixed with blood and mucus, and gradual onset. They also may have signs of weight loss, and blood on rectal examination. The disease is usually accompanied with different degrees of abdominal pain, from mild discomfort to severely painful cramps. Ulcerative colitis is believed to affect the entire body that leads to many symptoms outside the intestine. The effect of ulcerative colitis on mortality is unclear, but it is thought that the disease primarily affects quality of life, and not lifespan.
Classification and external resources
Endoscopic image of a sigmoid colon afflicted with ulcerative colitis
Ulcerative colitis occurs in approximately 1 in 1000 in the United States. Ulcerative colitis seems to have no known cause. The disease may be triggered in a susceptible person by environmental stresses. Dietary modification may reduce the discomfort of a person with the disease. Although ulcerative colitis is treated as though it were an autoimmune disease, there is no consensus that it is such. Surgical removal of the large intestine often necessary.
Ulcerative colitis is normally continuous from the rectum up the colon. Colitis is potentially manageable with enemas. Occasionally the ileum is involved.
Severity of disease
• Mild disease: Fewer than four stools daily, with or without blood, no systemic signs of toxicity, and a normal erythrocyte sedimentation rate (ESR). There may be mild abdominal pain or cramping. Patients may believe they are constipated when in fact they are experiencing tenesmus, which is a constant feeling of the need to empty the bowel accompanied by involuntary straining efforts, pain, and cramping with little or no fecal output. Rectal pain is uncommon.
• Moderate disease: More than four stools daily, but with minimal signs of toxicity. Patients may display anemia, moderate abdominal pain, and low grade fever 99.5 to 102.2 °F.
• Severe disease: More than six bloody stools a day, and evidence of toxicity as demonstrated by fever, racing heart rate, anemia or an elevated ESR.
• Fulminant disease: More than ten bowel movements daily, continuous bleeding, toxicity, abdominal tenderness and distension, blood transfusion requirement and colonic expansion. Patients in this category may have inflammation extending beyond just the mucosal layer, causing impaired colonic motility and leading to toxic megacolon or colonic perforation may ensue. Unless treated, fulminant disease will soon lead to death.
Many hypotheses have been raised for environmental contributants to the pathogenesis of ulcerative colitis. They include the following:
• Diet: as the colon is exposed many environmental toxins from what we eat and drink which may promote inflammation in ulcerative colitis and Crohn's disease.
• Diet: A diet low in fermentable dietary fiber may affect ulcerative colitis incidence and food allergies.
• Breastfeeding: There have been conflicting reports of the protection of breastfeeding in the development of inflammatory bowel disease. One Italian study showed a potential protective effect.
• Several scientific studies have posted that Accutane is a possible trigger of Crohn's Disease and Ulcerative colitis in some individuals. Three cases in the United States have gone to trial thus far, with all three resulting in multi-million dollar judgements against the makers of isotretinoin; there are an additional 425 cases pending.
Ulcerative colitis is believed to have a systemic (i.e., autoimmune) origin, patients may present with complications outside the colon. These include the following:
Patients with ulcerative colitis can occasionally have aphthous ulcers involving the tongue, lips and throat
• aphthous ulcers of the mouth
o Iritis or uveitis, which is inflammation of the iris.
• Inflammatory Arthritis of the feet, hands and spine, including ankylosing spondylitis
o Erythema nodosum, or inflammation of subcutaneous tissue involving the lower extremities
o Pyoderma gangrenosum, which is a painful ulcerating lesion involving the skin
• pulmonary embolism
• Autoimmune hemolytic anemia
• clubbing, a deformity of the ends of the fingers
• Primary sclerosing cholangitis, a distinct disease that causes inflammation of the bile ducts
The following conditions may present in a similar manner as ulcerative colitis, and should be excluded:
• Crohn's Disease
• Infectious colitis, which is typically detected on stool cultures
• Pseudomembranous colitis, or Clostridium difficile-associated colitis, bacterial upsets often seen following administration of antibiotics
• Ischemic colitis, inadequate blood supply to the intestine, which typically affects the elderly
• Radiation colitis in patients with previous pelvic radiotherapy.
• Chemical colitis resulting from introduction of harsh chemicals into the colon from an enema or other procedure.
Ulcerative Colitis comparison to Crohn's Disease
The most common disease that mimics the symptoms of ulcerative colitis is Crohn's Disease, as both are inflammatory bowel diseases that can affect the colon with similar symptoms. It is important to differentiate these diseases, since the course of the diseases and treatments may be different. In some cases, however, it may not be possible to tell the difference, in which case the disease is classified as indeterminate colitis.
Comparisons of various factors in Crohn's disease and ulcerative colitis
Crohn's Disease Ulcerative Colitis
Involves terminal ileum? Commonly Seldom
Involves colon? Usually Always
Involves rectum? Seldom Usually
Peri-anal involvement? Commonly Seldom
Bile duct involvement? Not associated Higher rate of Primary sclerosing cholangitis
Distribution of Disease Patchy areas of inflammation Continuous area of inflammation
Endoscopy Linear and serpiginous (snake-like) ulcers Continuous ulcer
Depth of inflammation May be transmural, deep into tissues Shallow, mucosal
Fistulae, abnormal passageways between organs Commonly Seldom
Biopsy Can have granulomata Crypt abscesses and cryptitis
Surgical cure? Often returns following removal of affected part Usually cured by removal of colon, can be followed by pouchitis
Smoking Higher risk for smokers Lower risk for smokers
Autoimmune disease? Generally regarded as an autoimmune disease No consensus
Cancer risk? Lower than ulcerative colitis Higher than Crohn's
General Work-Up Of The Condition
H&E stain of a colonic biopsy showing a crypt abscess, a classic finding in ulcerative colitis
The initial diagnostic workup for ulcerative colitis includes the following:
• A complete blood count is done to check for anemia; thrombocytosis, a high platelet count, is occasionally seen
• Electrolyte studies and renal function tests are done, as chronic diarrhea may be associated with hypokalemia, hypomagnesemia and pre-renal failure.
• Liver function tests are performed to screen for bile duct involvement: primary sclerosing cholangitis.
• Stool culture, to rule out parasites and infectious causes.
• Erythrocyte sedimentation rate can be measured, with an elevated sedimentation rate indicating that an inflammatory process is present.
• C-reactive protein can be measured, with an elevated level being another indication of inflammation.
The best test for diagnosis of ulcerative colitis remains endoscopy. Full colonoscopy to the cecum and entry into the terminal ileum is attempted only if diagnosis of UC is unclear.
• Loss of the vascular appearance of the colon
• Erythema (or redness of the mucosa) and friability of the mucosa
• Superficial ulceration, which may be confluent, and
Biopsies of the mucosa are taken to definitively diagnose UC and differentiate it from Crohn's disease, which is managed differently clinically.
Course of Condition and It’s Complications:
Progression or Remission
Patients with ulcerative colitis usually have an intermittent course, with periods of disease inactivity alternating with "flares" of disease. Patients with more extensive disease are less likely to sustain remission, but the rate of remission is independent of the severity of disease.
Ulcerative Colitis and Colorectal Cancer
There is a significantly increased risk of colorectal cancer in patients with ulcerative colitis after 10 years. It is recommended that patients have screening colonoscopies with random biopsies to look for dysplasia after eight years of disease activity.
Primary Sclerosing Cholangitis
Ulcerative colitis has a significant association with primary sclerosing cholangitis (PSC), anti-inflammatory disorder of the bile duct. As many as 5% of patients with ulcerative colitis may progress to develop primary sclerosing cholangitis.
Natural support for Ulcerative Colitis
Unlike Crohn's disease, ulcerative colitis has a lesser prevalence in smokers than non-smokers.
Dietary fiber, meaning indigestible plant matter, has been recommended for decades in the maintenance of bowel function. Of peculiar note is fiber from brassica, which seems to contain soluble constituents capable of reversing ulcers along the entire human digestive tract before it is cooked. Oatmeal is also commonly prescribed.
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Crohn’s disease is classified as autoimmune disease, caused by one’s own immune system attacking the gastrointestinal track and producing inflammation. Thus, also classified as inflammatory bowel disease and may affect any part of the gastrointestinal tract from mouth to anus, causing a wide variety of symptoms.
50% of the time Crohn’s disease affects the ileum (the last part of the small intestine that connects to the large intestine. For this reason, the disease has also been called regional ileitis or regional enteritis.
There appears to be a genetic link to Crohn's disease, with the highest risk occurring in individuals with siblings with the disease. They’re up to 30 times more likely to develop it than the normal population. Also, smokers are three times more likely to develop Crohn's disease. Crohn's disease affects between 400,000 and 600,000 people in North America and can occur at any age.
Many people with Crohn's disease have symptoms for years prior to their diagnosis. Abdominal pain may be the initial symptom of Crohn's disease. It is often accompanied by bloating, flatulence (gassy), diarrhea (which may be bloody), vomiting, weight loss (feel better when not eating or malabsorption of food), and an increase frequency of bowel movements (in some cases more than 20 bowel movements per day). Crohn’s disease may also cause complications outside of the gastrointestinal tract such as skin rashes, arthritis and inflammation of the eye.
The Three categories of Crohn’s Disease
1. Stricturing disease causes narrowing of the intestine which may lead to bowel obstruction or blockage in the bowel movement.
2. Penetrating disease creates abnormal passageways (fistulae) between the bowel and other structures such as the skin.
3. Inflammatory disease causes inflammation without causing strictures or fistulae.
Complications with Crohn’s Disease:
Itchiness or pain around the anus may be suggestive of inflammation, fistulization, fissure or abscess around the anal area. Perianal skin tags are also common in Crohn's disease. At the opposite end of the gastrointestinal tract, the mouth may be affected by non-healing sores (aphthous ulcers). Rarely, the esophagus and stomach are involved in Crohn's disease.
Crohn's disease can also cause neurological complications in up to 15% of patients). The most common of these are seizures, stroke, myopathy, peripheral neuropathy, headache and depression.
Crohn's patients often also have issues with Small bowel bacterial overgrowth syndrome, which has similar symptoms.
There is no known drug or surgical cure for Crohn's disease; Western treatment options are restricted to managing symptoms, and attempting to maintaining remission and preventing relapse.
The immunodeficiency, which has been shown to be due to (at least in part) impaired cytokine secretion by macrophages, is thought to lead to a sustained microbial-induced inflammatory response, particularly in the colon where the bacterial load is especially high. Crohn's disease is thought to be an autoimmune disease, with inflammation stimulated by an over-active Th1 cytokine response. However, more recent evidence has shown that Th17 is of greater importance in the disease. The most recent gene to be implicated in Crohn's disease is ATG16L1, which may reduce the effectiveness of autophagy, and hinder the body's ability to attack invasive bacteria.
It’s believe that a variety of microorganisms (like unhealthy bacteria, yeast, parasites, and virus) will grow due to the damage to the intestinal wall from crohn’s and add to the symptoms and further failure of one’s health. Some studies have suggested that Mycobacterium avium subsp. paratuberculosis plays a role in Crohn's disease, in part because it causes a very similar disease, Johne's disease, in cattle.
A Colonoscopy is one of the best test in diagnosing Crohn’s disease, it allows visualization of the colon and terminal ileum and is 70% effective. Though several other tests are required to confirm the diagnosis and direction of therapy.
CT and MRI scans are useful for evaluating the small bowel with enteroclysis protocols. They are additionally useful for looking for intra-abdominal complications of Crohn's disease such as abscesses, small bowel obstruction, or fistulae.
Blood tests can reveal anemia, which may be caused either by blood loss or vitamin B12 deficiency (associated with malabsorption with ileitis), or anaemia of chronic disease, characterized by its microcytic and hypochromic anaemia. . Erythrocyte sedimentation rate, or ESR, and C-reactive protein measurements can also be useful to measure the degree of inflammation.
Testing for anti-Saccharomyces cerevisiae antibodies (ASCA) and anti-neutrophil cytoplasmic antibodies (ANCA) has been evaluated to identify inflammatory diseases of the intestine and to differentiate Crohn's disease from ulcerative colitis.
Calprotectin is a protein found in neutrophil cytosol. Neutrophils are integral to the inflammatory process caused by Crohn's disease. Faecal Calprotectin is a protein measured in a patient's feces. The fecal concentration correlates well with inflammation and disease activity. Normal fecal calprotectin level in a patient with active gastrointestinal symptoms excludes inflammatory bowel disease as a likely diagnosis and in many cases negates the need for colonoscopy or radio labelled white cell scanning.
Comparison w/ Ulcerative Colitis
The most common disease that mimics the symptoms of Crohn's disease is ulcerative colitis, as both are inflammatory bowel diseases that can affect the colon with similar symptoms. It is important to differentiate these diseases, since the course of the diseases and treatments may be different. In some cases, however, it may not be possible to tell the difference, in which case the disease is classified as indeterminate colitis.
Comparisons of various factors in Crohn's disease and ulcerative colitis
Crohn's disease Ulcerative colitis
Terminal ileum involvement Commonly Seldom
Colon involvement Usually Always
Rectum involvement Seldom Usually
Involvement around the anus Common Seldom
Bile duct involvement No increase in rate of primary sclerosing cholangitis Higher rate
Distribution of Disease Patchy areas of inflammation (Skip lesions) Continuous area of inflammation
Endoscopy Deep geographic and serpiginous (snake-like) ulcers Continuous ulcer
Depth of inflammation May be transmural, deep into tissues Shallow, mucosal
Fistulae Common Seldom
Stenosis Common Seldom
Autoimmune disease Widely regarded as an autoimmune disease No consensus
Cytokine response Associated with Th17 Vaguely associated with Th2
Granulomas on biopsy Can have granulomas Granulomas not seen
Surgical cure Often returns following removal of affected part Usually cured by removal of colon
Smoking Higher risk for smokers Lower risk for smokers
A recent small double-blind, placebo-controlled study conducted in Germany found grand wormwood to have a steroid-sparing effect with the result that 18/20 wormwood receiving patients were able to taper steroid medication while maintaining a steady improvement in CD symptoms.
Recent research in France has suggested that a shortage of the bacterium Faecalibacterium prausnitzii may cause Crohn's disease by overstimulating the immune system. The researchers said that if ongoing animal trials prove successful, human patients could benefit from a probiotic treatment with F. prausnitzii. However, it was stated "It is too early to tell whether this will lead directly to a new treatment as other probiotics have tended to produce good results in animal studies only to prove disappointing in clinical trial in Crohn's disease."
At The Get Well Center we offer many natural approaches to help the body help itself, and help you live a better life.
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A peptic ulcer or peptic ulcer disease, is an erosion (resembling a crater) greater than 0.5 cm to the mucosal lining of the gastrointestinal track. As many as 80% of ulcers are associated with Helicobacter pylori, a spiral-shaped bacterium that lives in the acidic environment of the stomach, however only 20% of those cases go to a doctor. Peptic Ulcers can also be caused or worsened by drugs such as aspirin and other nonsteroidal anti-inflammatory agents or NSAIDs.
Contrary to general belief, more peptic ulcers arise in the duodenum (first part of the small intestine, just after the stomach) than in the stomach. About 4% of stomach ulcers are caused by a cancer.
Peptic ulcer may arise at various locations:
• Stomach (called gastric ulcer)
• Duodenum (called duodenal ulcer)
• Oesophagus (called Oesophageal ulcer)
• Meckel's Diverticulum (called Meckel's Diverticulum ulcer)
Types of peptic ulcers:
• Type I: Ulcer along the lesser curve of stomach
• Type II: Two ulcers present - one gastric, one duodenal
• Type III: Prepyloric ulcer
• Type IV: Proximal gastroesophageal ulcer
• Type V: Anywhere along gastric body, NSAID induced
Signs and symptoms of Peptic Ulcer
• abdominal pain, classically epigastric with severity relating to mealtimes, duodenal ulcers are classically relieved by food, while gastric ulcers are exacerbated by it
• bloating and abdominal fullness;
• waterbrash (rush of saliva after an episode of regurgitation to dilute the acid in esophagus);
• nausea, and lots of vomiting;
• loss of appetite and weight loss;
• hematemesis (vomiting of blood); this can occur due to bleeding directly from a gastric ulcer, or from damage to the Oesophagus from severe/continuing vomiting.
• melena (tarry, foul-smelling feces due to oxidized iron from hemoglobin)
• rarely, an ulcer can lead to a gastric or duodenal perforation. This is extremely painful and requires immediate surgery.
A history of heartburn, gastroesophageal reflux disease (GERD) and use of certain forms of medication can raise the suspicion for peptic ulcer. Medicines associated with peptic ulcer include NSAID (non-steroid anti-inflammatory drugs), and most glucocorticoids (e.g. dexamethasone and prednisolone).
The timing of the symptoms in relation to the meal may differentiate between gastric and duodenal ulcers: A gastric ulcer would give epigastric pain during the meal, as gastric acid is secreted, or after the meal, as the alkaline duodenal contents reflux into the stomach. Symptoms of duodenal ulcers would manifest mostly before the meal—when acid (production stimulated by hunger) is passed into the duodenum. However, this is not a reliable sign for certain with differential diagnosis.
• Gastrointestinal bleeding is the most common complication. Sudden large bleeding can be life-threatening. It occurs when the ulcer erodes one of the blood vessels.
• Perforation (a hole in the wall of the intestine) often leads to catastrophic consequences. Ulcer erosion into the lining of the intestine leads to leaking stomach or intestinal content into the abdominal cavity. Perforation at the front of the stomach leads to acute peritonitis, a life-threatening abdominal infection initially. The first sign is often sudden intense abdominal pain. Back wall perforation leads to pancreatitis; pain in this situation often radiates to the back.
• Penetration is when the ulcer continues into adjacent organs such as the liver and pancreas.
• Scarring and swelling due to ulcers causes narrowing in the duodenum and gastric outlet obstruction (food can’t pass into the small intestine) Patient often presents with severe vomiting.
• Pyloric stenosis narrowing of the lower esophagus. One experiences frequent acid reflux or heartburn.
Tobacco smoking, not eating properly, blood group, spices and other factors that were suspected to cause ulcers until late in the 20th century, are actually of relatively minor importance in the development of peptic ulcers.
A major causative factor (60% of gastric and up to 90% of duodenal ulcers) is chronic inflammation due to Helicobacter pylori bacteria that infects the gut. This bacteria causes chronic active gastritis, that usually decrease in gastrin production by the stomach, resulting in hypo- or achlorhydria. But it can occasionally increase gastrin stimulates the production of gastric acid (HCL), that can contribute to the erosion of the intestinal lining, and therefore ulcer formation. Studies have shown eating cabbage or cabbage juice can increase the mucosa lining in the stomach.
Another major cause is the common use of NSAIDs (drugs used for pain and inflammation). The incidence of duodenal ulcers has dropped significantly during the last 30 years, while the incidence of gastric ulcers has shown a small increase, mainly caused by the widespread use of NSAIDs.
Mental and Emotional Stress can play a role in the development of peptic ulcers. Despite the finding that a bacterial infection (H-pylori) is the cause of ulcers in 80% of cases, bacterial infection does not appear to explain all ulcers and researchers continue to look at stress as a possible cause, or at least a complication in the development of ulcers. An expert panel convened by the Academy of Behavioral Medicine Research concluded that ulcers are not purely an infectious disease and that psychological factors do play a significant role. Stress may also promote H. pylori infection ulcers because. Helicobacter pylori thrives in an acidic environment, and stress is associated to the production of excess stomach acid.
A study of peptic ulcer patients in a Thai hospital showed that chronic stress was strongly associated with an increased risk of peptic ulcer, and a combination of chronic stress and irregular mealtimes was a significant risk factor.
Smoking leads to atherosclerosis and vascular spasms, causing vascular insufficiency and promoting the development of ulcers through ischemia. Nicotine contained in cigarettes can increase parasympathetic nerve activity to the gastrointestinal tract by acting on the nicotinic receptors and increase the production of histamine and gastrin secreted and therefore increases the acidity and inflammation in the stomach and intestine.
There is an increased risk of duodenal ulcers when there is a family history of ulcers, especially when blood group O is also present. Inheritance appears to be unimportant in gastric ulcers.
Differential diagnosis of epigastric (stomach) pain
• Peptic ulcer
• Stomach cancer
• Gastroesophageal reflux disease
• Hepatic congestion
• Biliary colic
• Inferior myocardial infarction
• Referred pain (pleurisy, pericarditis)
Gastroscopy can directly visually identify the presents, location and severity of an ulcer.
The diagnosis of Helicobacter pylori can be made by:
• Urea breath test
• Direct detection of urease activity in a biopsy specimen by rapid urease test;
• Measurement of antibody levels in blood.
• Stool antigen test;
• Histological examination and staining of an EGD biopsy to in certain cases to rule out cancer.
Younger patients with ulcer-like symptoms are often treated with antacids or H2 antagonists before EGD is undertaken but, this doesn’t treat the cause of their symptoms.
Treatment of H. pylori usually leads to clearing of infection, relief of symptoms and eventual healing of ulcers. But this doesn’t treat other possible disorders lurking in or out of the gut. Full evaluation should be made to determine other possible digestive conditions.
Studies have shown eating cabbage or cabbage juice can increase the mucosa lining in the stomach.
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Most people with diverticulosis do not have any discomfort or symptoms; however, symptoms may include mild cramps, bloating, and constipation. Other diseases such as irritable bowel syndrome (IBS) and peptic ulcers cause similar problems, so these symptoms do not always mean a person has diverticulosis.
The differential diagnosis includes colon cancer, inflammatory bowel disease, colitis, and irritable bowel syndrome, as well as a number of urological and gynecological processes.
Patients with the above symptoms are commonly studied with a computed tomography, or CT scan. The CT scan is very sensitive (98%) in diagnosing diverticulitis. In order to extract the most information possible about the patient's condition, thin section (5mm) transverse images are obtained through the entire abdomen and pelvis after the patient has been administered oral and intravascular contrast. Images reveal localized thickening and hyperemia (increased blood flow) involving a segment of the colon wall, with inflammatory changes extending into the fatty tissues surrounding the colon. The diagnosis of acute diverticulitis is made confidently when the involved segment contains diverticula. CT may also identify patients with more complicated diverticulitis, such as those with an associated abscess. It may even allow for radiologically guided drainage of an associated abscess, sparing a patient from immediate surgical intervention.
Other studies, such as barium enema and colonoscopy are contraindicated in the acute phase of diverticulitis due to the risk of perforation.
Upon discharge patients may be placed on a low residue diet. This low-fiber diet gives the colon adequate time to heal without needing to be overworked. Later, patients are placed on a high-fiber diet. There is some evidence this lowers the recurrence rate.
In some cases surgery may be required to remove the area of the colon with the diverticula. Patients suffering their first attack of diverticulitis are typically not encouraged to undergo the surgery, unless the case is severe. Patients suffering repeated episodes may benefit from the surgery. In such cases the risks of complications from the diverticulitis outweigh the risks of complications from surgery.
Most cases of simple, uncomplicated diverticulitis respond to conservative therapy with bowel rest and antibiotics.
There is no scientific evidence that suggests the avoidance of nuts and seeds prevents the progression of diverticulosis to an acute case of diverticulitis, and as such the widely held belief that small indigestible foods like seeds becoming lodged in the diverticula appears to be nothing more than an 'old wives' tale. Further, in a survey of fellows of The American Society of Colon and Rectal Surgeons, although the majority of the surgeons responding to the survey favored adherence to a low residue diet, half of them still saw no value in specifically avoiding seeds and nuts.
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Diverticulitis is where diverticulosis (Pouching diverter fold of the colon lining) becomes inflamed. This can lead to the colon becoming infected with craters of food stuck inside, which causes abdominal pain.
Diverticulitis and Diverticulosis develops with raised diverticular colonic pressures due to improper motility of the gastric contents. The Sigmoid colon (95% of cases) has the smallest diameter of any portion of the colon, and therefore the portion which would be expected to have the highest intestinal pressure. The assumption that a lack of dietary fiber, particularly non-soluble fiber "roughage" predisposes individuals to diverticular disease is supported within the medical literature. Nut, corn and popcorn consumption has not been proven to increase the risk of diverticulosis or diverticular complications. There is some evidence that a genetic component may be a causative factor.
Large bowel (sigmoid colon) showing multiple diverticula. Note how the diverticula appear on either side of the longitudinal muscle bundle (taenium). There is some evidence that a genetic component may be a causative factor.
In complicated diverticulitis, bacteria may subsequently infect the outside of the colon if an inflamed diverticulum bursts open. If the infection spreads to the lining of the abdominal cavity, (peritoneum), this can cause a potentially fatal peritonitis. Sometimes inflamed diverticula can cause narrowing of the bowel, leading to an obstruction. It is thought that mechanical blockage of a diverticulum, possibly by a piece of feces or food particles, leads to infection of the diverticulum.
• Bowel obstruction
The most common symptom of diverticulitis is abdominal pain. The most common sign is tenderness around the left side of the lower abdomen. Other complaints are nausea or diarrhea. If diverticulitis becomes infection, then nausea, vomiting, feeling hot while having no fever, cramping, and constipation may occur as well. The severity of symptoms depends on the extent of the infection and complications. Diverticulitis worsens throughout the day, as it starts as small pains and slowly turns into vomiting and sharp pains.
On the blood study you can find an elevation of the white cell count (Leukocytosis).
In rare cases the tenderness can be the right side of the lower abdomen. This may appear to be appendicitis and only tests including a CT Scan will confirm it.
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Colorectal polyp or Colon polyps
A polyp is an abnormal growth of tissue projecting from an inner lining of the colon or rectum. If it is attached to the surface by a narrow-elongated stalk it is said to be pedunculated. Colon polyps are not usually associated with symptoms. Occasionally rectal bleeding, and on rare occasions pain, diarrhea or constipation may occur because of colon polyps. Colon polyps are a concern because of the potential for colon cancer being present microscopically and the risk of benign colon polyps transforming over time into malignant ones. Since most polyps are asymptomatic, they are usually discovered at the time of colon cancer screening with either digital rectal exam (DRE), flexible sigmoidoscopy, Barium enema, colonoscopy or virtual colonoscopy. The polyps are routinely removed at the time of colonoscopy either with a polypectomy snare or with biopsy forceps. Even though colon cancer is usually not found in polyps smaller than 2.5 cm, all polyps found are removed since the removal of polyps reduces the future likelihood of developing colon cancer. When adenomatous polyps are removed, a repeat colonoscopy is usually performed in three to five years
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Yeast or Candida (Candidiasis):
• What is Candida or Candidiasis?
• What The Get Well Center can do?
• What causes Candida?
• Leaky Gut Syndrome and Candidiasis
• Importance of diet
• Foods to avoid
• Foods to eat
What is Candida or Candidiasis?
Candida is a group of toxic, yeast-like microorganisms (Candida albicans being the most common) that can normally be found in the body (mouth, throat, intestine, bowel, genital/urinary tract) at low levels. In 20% of Americans, this toxic yeast overpopulates in the body resulting in deterioration of their health.
Overpopulation of yeast displays itself with various health complaints. Common examples are: Vaginal yeast infection (75% of females); gastrointestinal or digestive tract problems (constipation, diarrhea, colitis, abdominal pain, gas, distension, and heartburn); fatigue; mental issues; oral thrush; conjunctivitis; athlete’s foot; jock itch; diaper rash; muscle and joint pain; canker sores; sore throat; tingling sensations; kidney and bladder infections; infections of the nail, rectum, and other skin folds; depression; and even yeast complicated diabetes. The extent of problems related to candida overgrowth is apparent, but unfortunately candida is often not tested as a cause with most doctors. Diagnosis of yeast is important in order to treat the root causes instead of simply treating symptoms.
The Get Well Center we strive for success:
• Confirm the diagnosis of yeast and its severity.
• Determine if any other health issues are involved. “Knowledge is Power”
• Prescribe for you the best yeast cleansing agents.
• Repopulate your intestine with good bacteria and yeast to snuff out toxic yeast and bacteria, preventing the return of Candida.
• Provide you with an Anti-Candida diet to eliminate yeast and prevent the future reoccurrence of yeast.
Causes of Candida
A poor diet, an out-of-balance intestinal bacterial environment, and weakened immunity can all give rise to candida. Overuse of antibiotics, corticosteroids and oral contraceptives destroys the natural flora in the intestines that keep the candida cells in check. Carbohydrate rich diets heighten fertility of the body for yeast growth. Extreme stress, cancer patients undergoing chemotherapy, AIDS patients, infants, diabetics and others with weakened immune systems are more susceptible to an infection of candida (candidiasis).
Leaky Gut Syndrome and Candidiasis
In chronic Candidiasis, the rapidly multiplying candida can change its form and produce hook-shaped barbed appendages (Rhizoids) that cut into tissue and membranes, causing pain and other harmful symptoms. If not treated, a condition called leaky gut syndrome can develop in the intestinal track. Leaky Gut Syndrome allows microbes and undigested food to enter directly into the bloodstream. The result is toxic syndrome or even autoimmune disease.
Note, once someone begins treatment to kill the excess candida growth, the initial reaction to treatment will often result with the patient feeling worse! This is because the fungal form of candida does not want to die, and it fights like heck to survive!
Keeping Candida in Check Though Diet
A diet to eliminate Candida is a long-term lifestyle. Yeast loves a diet of sugar and simple carbohydrates, if you keep feeding yeast organisms they will multiply. Even the best cleansing agents won’t keep them in check without proper diet. At The Get Well Center we recommend eating a disciplined diet for at least 3 months along with other recommendations. Then, only after your symptoms disappear, you can expand your diet. Beware not to revert to old dietary habits or the candida symptoms will quickly return.
Foods to Avoid
Individual recovery times differ, but as a rule many Candida sufferers report that you must be on the diet for one month for every year which you have had Candida. That means, if you have had Candida for 27 years, then you need to follow the diet for twenty-seven months.
1. All Types of Sugar
Brown or white sugar, molasses, turbinado, demerara, honey, corn syrup, maple syrup, maple sugar, date sugar, amazake, rice syrup, sorghum.
Hidden sugars found in processed foods found on the label as: sucrose, dextrose, maltose, lactose, glycogen, glucose, mannitol, sorbitol, galactose, monosaccharides, and polysaccharides.
Fruit sugars (fructose) also feed Candida, so avoid all frozen, canned, and dried fruit, and fruit juices.
You can squeeze lemon or lime juice on your food, or in a glass of water.
Alcohol is high in sugar, sugar that can feed the growth of the Candida. Avoid alcoholic drinks like wine, beer, cider, whiskey, brandy, gin, scotch, any fermented liquor, vodka, rum, and all liqueurs.
Baked foods (for example, breads, rolls, crackers, bagels, pastries, muffins, cakes, and cookies) contain high levels of sugars (carbohydrates).
4. Ready to eat cereals (excepting Shredded Wheat and Kashi)
5. Pasta or Noodles
Noodles are made with flour which contains significant amount of sugar. All types of noodles are made from the same base and they should all be cut out of the diet, for example Saifun (Japanese noodles), Ramen instant noodles, farina, semolina and white flour noodles and pastas.
6. Dairy Products
Avoid cow's milk, (whole, skim, 2% fat) and dry powdered milk. Dairy intake largely depends on the level of patient sensitivity to cow's milk and cow's milk products such as cheese, yogurt and butter.
7. Fungi and mold containing foods
Avoid mushrooms, peanuts, peanut butter, pistachios, melons (e.g. cantaloupe); moldy and/or processed cheese like Roquefort, stilton, blue cheeses; processed cheeses like cheese slices, Velveeta, cream cheese, cheese snacks.
Condiments tend to be high in sugar or vinegar and can exacerbate your Candida. Stay away from pickles, ketchup, mayonnaise, mustard, relish, steak sauce, horseradish, Worcestershire and soy sauce. Salad dressings, whether a sweet honey mustard sauce or a simple balsamic vinegar dressing, are best avoided. Looking for a substitute? Try a simple olive oil and lemon juice dressing.
9. Processed / Packaged Foods with restricted ingredients
Many processed, packaged, boxed, and bottled foods contain yeast, refined sugar, refined flour and/or other ingredients that encourage Candida. Examples include: sodas, potato chips, tortilla chips, pretzels, junk food.
Foods to Eat
It is evident that in order to maintain a healthy body free of an overgrowth of Candida, the foods that are eaten must be foods that promote healthy Candida levels in the body.
The ideal diet for those suffering from Candida yeast overgrowth is to eat a diet high in fiber and protein complemented by some complex carbohydrates and a small amount of fresh fruits. For example, the daily intake should be approximately:
65% high fiber foods, such as steamed vegetables: The best choices are broccoli, celery, radishes, asparagus, onions, garlic, ginger root, cabbage, turnips and kale. Raw garlic and onions act as natural anti-fungal substances and should be eaten as much as possible.
20% high protein foods, such as: fish, chicken, duck, nuts, seeds, and eggs.
10% complex carbohydrates: rice, beans and oats.
5% fresh fruits, such as: berries of all kinds, grapefruit, pineapple, and papaya.
The following foods when taken in the recommended daily portions will strengthen your immune system and promote a healthy body free of candida.
GRAINS: in moderation Whole Amaranth and flour; Whole Barley flour and hatomugi; Buckwheat flour and groats; Under germinated Corn meal (fine or coarse grind); Masa Harina; Popcorn (air popper), Whole Millet and flour; Oat bran, flour, Groats, Quick; whole Quinoa and flour; Wild Rice; Brown Rice (long, med. or short grains), Brown Basmati Rice, Texmati Brown Rice, Wehani Brown Rice Rye flour, Groats, Cream of rye cereal; Whole Spelt and flour; Whole Teff and flour; Wheat Berries, Unprocessed or miller's wheat bran, Bulgur, cracked, Durum, Graham wheat flour, Whole wheat flour. Corn, Quinoa, Saifun (Japanese noodles), Soba (buckwheat), Udon (Japanese noodles), Whole wheat.
Acceptable BAKED/PREPARED PRODUCTS: Any whole grain unsweetened, non-yeasted bread product such as chapattis, whole wheat flour, corn chips, quick breads, unsweetened rice cakes or crackers, tortillas made from brown, corn or whole wheat. Ryvita contains no yeast.
LEGUMES: Dried or frozen black-eyed peas, chickpeas or garbanzo beans, lentils, soybeans, split peas
Acceptable DAIRY: Unsweetened organic soymilk and almond milk, plain organic yogurt with added acidophilus culture.
NUTS & SEEDS: Almonds, Brazil, Cashews, Hazel, Macadamia, Pecans, Pine nuts, Poppy, Pumpkin, Sesame (tahini), Sunflower.
EAT VEGETABLES: Beans, Broccoli, Brussels sprouts Cabbage, Cauliflower, Celery, Cucumbers Green pepper, Kale, Lettuce Onion, radishes, Parsley, Peas, Rocket or Arugula Spinach, Tomatoes, small occasional amounts of fresh lemon, lime, tomato, and eggplant.
CONDIMENTS AND SEASONINGS: Chicken broth without sweetener, Fresh garlic, Fresh ginger, Fresh herbs, Fresh onion, Pepper, Salt, Tamari (unfermented soy sauce), Raw organic apple cider vinegar
MEATS: Antelope, beef, buffalo, caribou, chicken, deer, duck, eggs, elk, all types of fish, frog legs, game hen, goat, goose, grouse (partridge), guinea fowl, moose, mutton, ostrich, peafowl, pheasant, pigeon (squab), pork, quail, and turkey.
BREWER’S YEASTS: are not harmful, as they do not colonize in the intestines.
Why Antibiotics Could Make Your Candida Worse:
Antibiotics are designed to eradicate the bad bacteria that cause certain diseases and infections. While antibiotics destroy bad bacteria and stop the spread of infection, they also destroy the good bacteria that help to keep the Candida in the body under control. Prolonged or strong doses of antibiotics can lead to an imbalance in gut flora, an imbalance that allows bad bacteria, yeasts and parasites to overgrow in the gut.
How can you protect yourself against developing Candidiasis from antibiotics?
Don't take antibiotic treatment unless it is absolutely necessary. Illnesses such as the common cold, flu, and sinus infections do not always require antibiotics unless they prolong for more than one week, cause irregular symptoms, or produce a high fever.
Many people believe that antibiotics are a fast way to get rid of common infections. This may be true but, those who take antibiotics for these illnesses do not allow their immune system the chance to fight off the infection and become stronger. Instead, they allow their body to become dependent on antibiotics. The downside to this thought process is eventually the invading microbes will develop a resistance to these medications and will require a higher dosage or stronger antibiotics the next time they become sick.
Talk to your doctor - Find out if you can take a lower dosage of antibiotics or contact us for complimentary care to enhance your immune system.
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Leaky Gut Syndrome
Inflammatory Bowel Disease
• Yeast overgrowth
• Intestinal Parasites
• flora (bacteria) imbalance or Dysbiosis